Part:BBa_K3718010
Demonstrative circuit of tool kit part 2
GFP is produced and growth rate is restored in the presence of caffeine molecule. This acts as a proof of concept in our project. This part contains the plasmid insert of component II (cloning) of our toolkit.
After the experiments of the Modular Receptor Platform (MRP) expression, and the transformation of the arcA and iclR genes into E. coli, which has its arcA and iclR knocked out before the transformation, have proven to be successful. We will use caffeine-sensitive MRP as a testing part to model the system. Caffeine will be used to homodimerize the receptors, and is expected to induce the expression of downstream genes. This model will be used to conduct three experiments to confirm the functionality of the tool. By cloning in an antibody VHH gene against caffeine and green fluorescent protein (GFP) as the desired gene to be expressed, we expect the platform is activated in the presence of caffeine to express GFP, arcA and iclR, which will result in an increase in growth rate and a visible green color under UV.
Sequence and Features
- 10COMPATIBLE WITH RFC[10]
- 12INCOMPATIBLE WITH RFC[12]Illegal NheI site found at 7
Illegal NheI site found at 30 - 21INCOMPATIBLE WITH RFC[21]Illegal BglII site found at 120
Illegal BglII site found at 994
Illegal BglII site found at 3417 - 23COMPATIBLE WITH RFC[23]
- 25INCOMPATIBLE WITH RFC[25]Illegal AgeI site found at 686
- 1000INCOMPATIBLE WITH RFC[1000]Illegal BsaI.rc site found at 1974
Reference
H. Waegeman, J. Beauprez, H. Moens, J. Maertens, M. De Mey, M. R. Foulquié-Moreno, J. J. Heijnen, D. Charlier, and W. Soetaert, “Effect of ICLR and ARCA knockouts on biomass formation and metabolic fluxes in escherichia coli K12 and its implications on understanding the metabolism of escherichia coli BL21 (DE3),” BMC Microbiology, vol. 11, no. 1, p. 70, 2011.
H.-J. Chang, P. Mayonove, A. Zavala, A. De Visch, P. Minard, M. Cohen-Gonsaud, and J. Bonnet, “A modular receptor platform to expand the sensing repertoire of bacteria,” ACS Synthetic Biology, vol. 7, no. 1, pp. 166–175, 2017.
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